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G Protein Signaling Pathways in Health and Disease Tao Hardback Volume 161

ÂŁ100.79

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Product Description

G Protein Signaling Pathways in Health and Disease

Contains cutting-edge reviews in molecular biology, including the latest on prions, viruses, bacteria and eukaryotes

Ya-Xiong Tao (Volume editor)

9780128167243

Hardback, published 4 February 2019

229 pages

22.9 x 15.2 x 2.1 cm, 0.43 kg

G Protein Signaling Pathways in Health and Disease, Volume 161 in the Progress in Molecular Biology and Translational Science series, provides informative and exciting monographs on a wide variety of research topics related to G Protein Signaling Pathways in Health and Disease. The series gives in-depth knowledge on the important molecular biological aspects of organismal physiology and function, along with insights on how this knowledge can be applied to understand and ameliorate human disease. This updated release covers Diseases associated with mutations in CNGA3, Mutations in arrestins, Diseases caused by mutations in GPR101, Diseases caused by mutations in lutropin receptor, and]

G Protein Signaling Pathways in Health and Disease Tao Hardback Volume 161

G Protein Signaling Pathways in Health and Disease

Contains cutting-edge reviews in molecular biology, including the latest on prions, viruses, bacteria and eukaryotes

Ya-Xiong Tao (Volume editor)

9780128167243

Hardback, published 4 February 2019

229 pages

22.9 x 15.2 x 2.1 cm, 0.43 kg

G Protein Signaling Pathways in Health and Disease, Volume 161 in the Progress in Molecular Biology and Translational Science series, provides informative and exciting monographs on a wide variety of research topics related to G Protein Signaling Pathways in Health and Disease. The series gives in-depth knowledge on the important molecular biological aspects of organismal physiology and function, along with insights on how this knowledge can be applied to understand and ameliorate human disease. This updated release covers Diseases associated with mutations in CNGA3, Mutations in arrestins, Diseases caused by mutations in GPR101, Diseases caused by mutations in lutropin receptor, and]

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